G & K Fitness Medics 

cholesterol is an essential element in the body. It’s in all cell membranes, and a major constituent of key components including several hormones and vitamin D (actually, vitamin D is a hormone, despite it being branded a ‘vitamin’).

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SO, how did all of our so called medical professionals get it so wrong?

They base their evidence on a study done around the the turn of the sentry preformed on rabbits.  Rabbits are vegetarians, (that means they do not consume meat (Fat)) and for some odd reason what these so called brilliant minds  cant seem to figure out is that  cholesterol is only found in animals (Meat) and a rabbits body is not equipped to handle   (fat) cholesterol consumption which lead to a build up of place (atherosclerosis) in the rabbits veins.  There have been several study’s that show that there is absolutely no correlation with  atherosclerosis in omnivores when they consume (fat) cholesterol

Then in the 1950’s Ancel Benjamin Keys, an American scientist who studied the influence of diet on health. In particular, he came up with The lipid hypothesis, which states that eating saturated fats raises cholesterol and clogs arteries causing heart disease  Ancel had access to reliable data from 22 countries.  But in order to get the results that he wanted he threw out numerous amounts of data that proved his theory wrong and only included the data from the 6 countries that would allow him to manipulate the results to what he wanted them to be.

So why wont they change health care practices?  Because they would have to admit being wrong,  Which doctors are incapable of?  Or maybe because of the huge monetary losses that the health care and pharmaceutical industries would realize when being honest with the American public?

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

THE FACTS!

A high blood cholesterol is said to promote atherosclerosis and thus also coronary heart disease. But many studies have shown that people whose blood cholesterol is low become just as atherosclerotic as people whose cholesterol is high. There is no relationship between the blood cholesterol level and the degree of atherosclerosis in the vessels.
 

1. Your body produces three to four times more cholesterol than you eat. The production of cholesterol increases when you eat little cholesterol and decreases when you eat much. This explains why diet cannot lower cholesterol more than on average a few per cent.
    

2. There is no evidence that too much animal fat and cholesterol in the diet promotes atherosclerosis or heart attacks. For instance, more than twenty studies have shown that people who have had a heart attack haven’t eaten more fat of any kind than other people, and degree of atherosclerosis at autopsy is unrelated with the diet.
    

3. The only effective way to lower cholesterol is with drugs, but neither heart mortality or total mortality have been improved with drugs the effect of which is cholesterol-lowering only. On the contrary, these drugs are dangerous to your health and may shorten your life.
    

4. The modern cholesterol-lowering drugs, the statins, do prevent cardiovascular disease, but the effect is minuscule and is due to other mechanisms than cholesterol-lowering. Unfortunately, they also stimulate cancer, disturb the functions of the muscles, the heart and the brain, create an immune system deficit and pregnant women taking statins may give birth to children with malformations more severe than those seen after thalidomide.

 

 

 

 

 

 

 

Effects of low Cholesterol on the immune system

cholesterol may have a role in the body’s ability to resist infection too. Some evidence for a protective role here comes from studies which link higher cholesterol levels with reduced risk of infection. In fact, we can add “immune system deficit” to the list of statin drug side effects since low cholesterol levels can make you more susceptible to infections.

 

In one study, for instance, over a 15-year period, those with higher initial levels of total cholesterol were found to be less likely to be admitted to a hospital with an infectious disease.

 

A cholesterol-rich diet accelerated the sterilization rate of sputum cultures in pulmonary tuberculosis patients, suggesting that cholesterol should be used as a complementary measure in antitubercular treatment.

A study of 21 individuals who had confirmed infection with tuberculosis (TB). All of the individuals were treated with standard TB medication (four antibiotics taken in combination) over a period of 8 weeks.

Of the 21 participants, 10 were given a cholesterol-rich diet (800 mg of cholesterol a day – about the amount of cholesterol found in 5 medium-sized eggs). The rest of the study participants were to eat a diet which contained 250 mg of cholesterol each day.

After two weeks of treatment, 80 per cent of those eating a high-cholesterol diet were free of TB infection, compared to only 9 per cent of the others. This difference was statistically significant. 

 

Study's suggest that the higher cholesterol diet may have benefitted individuals not through a rise in blood cholesterol levels, but by “[replenishing] metabolic pools (e.g. cell membranes), transformed into other products such as hormones and vitamins A and D, or catabolized through bile salts. Thus, it is reasonable to speculate that the replenishment of metabolic pools occurred faster in those patients eating a cholesterol-rich diet than in control subjects, even when no differences in serum cholesterol levels were observed between the groups due to the catabolism of cholesterol into bile acids.

 

• In 1997, researchers at the University of Pittsburgh’s Center for Clinical Pharmacology checked the immune systems of healthy adult men (average age 46) who had either high or low cholesterol. There were significantly fewer circulating white blood cells in the low cholesterol group, suggesting a less efficient immune system.

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• A 1998 investigation into the association between total cholesterol and risk of infections (other than respiratory and HIV) diagnosed within the California Kaiser Permanente system tracked some 55,000 men and 65,000 women for 15 years (1979–1993). What they found is that higher total cholesterol was more protective.

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• Higher cholesterol reduces the risk of respiratory infections, according to an earlier study by the same researchers as the above study, using slightly fewer cases. The risk was stronger among men, and in older compared to younger participants. Asthma was not related to cholesterol.

• Individuals with LDL cholesterol below 70 have 15 times the risk of developing cancer, and five times the risk of contracting sepsis (blood poisoning), according to a 2007 Israeli study of 203 patients. 

   

   

   

   

   

   

   

   

   

 

What is Cholesterol

Cholesterol is a peculiar molecule. It is often called a lipid or a fat. However, the chemical term for a molecule such as cholesterol is alcohol, although it doesn’t behave like alcohol. Its numerous carbon and hydrogen atoms are put together in an intricate three-dimensional network, impossible to dissolve in water. All living creatures use this indissolvability cleverly, incorporating cholesterol into their cell walls to make cells waterproof.This means that cells of living creatures can regulate their internal environment undisturbed by changes in their surroundings, a mechanism vital for proper function. The fact that cells are waterproof is especially critical for the normal functioning of nerves and nerve cells. Thus, the highest concentration of cholesterol in the body is found in the brain and other parts of the nervous system.

The submarines, or lipoproteins, have various names according to their density. The best known are HDL (High Density Lipoprotein), and LDL (Low Density Lipoprotein). The main task of HDL is to carry cholesterol from the peripheral tissues, including the artery walls, to the liver. Here it is excreted with the bile, or used for other purposes, for instance as a starting point for the manufacture of important hormones.The LDL submarines mainly transport cholesterol in the opposite direction. They carry it from the liver, where most of our body’s cholesterol is produced, to the peripheral tissues, including the vascular walls

 

 

Body weight, smoking, exercise…

 

People who reduce their body weight also reduce their cholesterol. In a review of 70 studies Dr. Anne Dattilo and Dr. P.M. Kris-Etherton concluded that, on average, weight reduction lowers cholesterol by about 10 per cent, depending on the degree of the reduction. Interestingly, it is only cholesterol transported by LDL that goes down; the small part transported by HDL goes up. In other words, weight reduction increases the ratio between HDL- and LDL-cholesterol. An increase of the HDL/LDL ratio is called ”favorable” by the diet-heart supporters; cholesterol is changed from ”bad” to ”good”. But is it the ratio or the weight reduction that is favorable? When we become fat, other harmful things occur to us. One is that our cells become less sensitive to insulin, so that some of us develop diabetes. And people with diabetes are much more likely to have a heart attack than people without diabetes, because atherosclerosis and other vascular damage occur very early in diabetics, even in those without lipid abnormalities. In other words, overweight may increase the risk of a heart attack by mechanisms other than an unfavorable lipid pattern, while at the same time overweight lowers the HDL/LDL ratio.

 

 

 

 

 

 

 

Smoking increases cholesterol a little. Again, it is LDL-cholesterol that increases, while HDL-cholesterol goes down, resulting in an ”unfavorable” HDL/LDL ratio. What is certainly unfavorable is the chronic exposure to the fumes from burning paper and tobacco leaves. Instead of considering the low HDL/LDL ratio as bad it could simply be smoking itself that is bad. Smoking may provoke a heart attack and, at the same time, lower the HDL/LDL ratio.

 

Exercise decreases the bad LDL-cholesterol and increases the ”good” HDL-cholesterol. In well-trained individuals the ”good” HDL is increased considerably. In a comparison between distance runners and sedentary individuals, Dr. Paul D. Thompson and his colleagues found that the athletes on average had a 41 per cent higher HDL-cholesterol level. Most population studies have shown that physical exercise is associated with a lower risk of coronary heart disease, and a sedentary life with a higher risk. It also seems plausible that a well-trained heart is better guarded against obstruction of the coronary vessels than a heart always working at low speed. A sedentary life may predispose people to a heart attack and, at the same time, lower the HDL/LDL ratio.

 

A low ratio is also associated with high blood pressure. Most probably, the hypertensive effect is created by the sympathetic nerve system, which is often overstimulated in hypertensive patients. Hypertension (or too much adrenalin) may provoke a heart attack, for instance by inducing spasm of the coronary arteries or by stimulating the arterial muscle cells to proliferate, and, at the same time, lower the HDL/LDL ratio.